Download Diabetes and Cardiovascular Disease by Aresh J. Anwar MD, MRCP, Anthony H. Barnett MD, FRCP, PDF

By Aresh J. Anwar MD, MRCP, Anthony H. Barnett MD, FRCP, Sudhesh Kumar MD, FRCP (auth.), Michael T. Johnstone MD, CM, FRCP(C), Aristidis Veves MD, DSC (eds.)

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05), representing a reduction of glucose uptake of about 25% (Fig. 4), well in line with what had been predicted according to the experimentally defined correlation coefficients. In a third series of studies, we examined whether rates of skeletal muscle glucose uptake in response to changes in leg blood flow followed a noncapillary recruitment model, as proposed by Renkin, or whether changes in glucose uptake were dependent on capillary recruitment. The results of this study revealed that leg glucose uptake in response to pharmacologic manipulation of blood flow were different from those predicted by the Renkin model (37a), indicating that capillary recruitment is important for the metabolic actions of insulin (38).

05), representing a reduction of glucose uptake of about 25% (Fig. 4), well in line with what had been predicted according to the experimentally defined correlation coefficients. In a third series of studies, we examined whether rates of skeletal muscle glucose uptake in response to changes in leg blood flow followed a noncapillary recruitment model, as proposed by Renkin, or whether changes in glucose uptake were dependent on capillary recruitment. The results of this study revealed that leg glucose uptake in response to pharmacologic manipulation of blood flow were different from those predicted by the Renkin model (37a), indicating that capillary recruitment is important for the metabolic actions of insulin (38).

Dresner and colleagues (66) demonstrated that insulin resistance induced by FFA elevation was associated with decreased PI3K activity in skeletal muscle. Therefore, if insulin signaling pathways are shared in endothelial cells and skeletal muscle, one may expect impaired insulin signaling in the endothelial cells in response to euglycernic hyperinsulinemia with superimposed FFA elevation. Other evidence for the ability of elevated FFA levels to reduce endothelial NO production comes from in vitro studies.

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