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Divided into 5 expansive sections, this consultant offers an in-depth overview of crystal-induced arthritis and similar issues. delivering a historic standpoint all through, this paintings contains designated descriptions of the scientific beneficial properties of those illnesses, in addition to a present review in their identity, epidemiology, genetics, and underlying pathophysiology. This authoritative advisor additionally reports case experiences, present diagnostic instruments, and the superiority of those stipulations whereas introducing new and rising remedies for affliction administration.
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Cet ouvrage traite de l’ensemble des manifestations cutan? es et muqueuses observ? es au cours des diff? rentes maladies dites syst? miques, communes ou rares. Chaque chapitre aborde de fa? on exhaustive et originale les connaissances cliniques, anatomopathologiques et th? rapeutiques indispensables ?
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Extra resources for Crystal-Induced Arthropathies: Gout, Pseudogout and Apatite-Associated Syndromes
Trentham DE, Masi AT, Hamm RL. Letter: Roentgenographic prevalence of chondrocalcinosis. Arthritis Rheum 1975; 18:627–628. 116. Ellman MH, Levin B. Chondrocalcinosis in elderly persons. Arthritis Rheum 1975; 18:43–47. 117. Leonard A, Solnica J, Cauvin M, Houdent G, Mallet E, Brunelle P, Deshayes P. [Chondrocalcinosis: study of its radiologic frequency and its relation to arthrosis. Study of the level of parathyroid homone]. Rev Rhum Mal Osteoartic 1977; 44:559–564. 118. Delauche MC, Stehle B, Cassou B, Verret JM, Kahn MF.
Studies, the NHANES and the Framingham study, have fueled controversy about whether serum urate is a causal factor in cardiovascular disease. For example, in NHANES I, a 60 micromol/L increase in serum urate level was associated with a 48% increase in the risk of ischemic heart disease among women (34). There was higher all-cause mortality and greater ethnic diversity in NHANES compared to Framingham, suggesting possible greater external validity (generalizability) of NHANES. On the other hand, critics contend that serum urate may simply be a confounder for hypertension, diuretic use, dyslipidemia, disordered glucose metabolism and/or renal insufficiency (95,98).
Investigators found differences in gout risk based on the type of alcohol consumed (Fig. 3). Gout risk appeared to be most strongly associated with beer intake, perhaps related to its greater purine content (guanosine) relative to other alcoholic beverages (53). The relative risk for each daily 12 oz. 28). In contrast, there was no association of daily wine consumption with gout risk (RR per 4 oz. 22). 16 Saag et al. 68 0 < 1/m 5-7/w >1/d < 1/m 5-7/w >1/d < 1/m 5-7/w >1/d Alcohol Types (serving/time) Figure 3 Gout risk based on the type of alcoholic beverage consumed.