By Raymond S Koff; George Y Wu
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Additional resources for Chronic viral hepatitis : diagnosis and therapeutics
Chronic hepatitis B virus infection may be divided into two phases: highly replicative and relatively nonreplicative. Perinatally acquired HBV infection appears to be associated with immunologic tolerance to the virus (postulated to be the result of in utero exposure to HBeAg, which functions as a tolerogen) (see Fig. 2a). This results in high levels of viral replication, without a robust immune response, and, therefore, minimal evidence of hepatic injury by histologic, biochemical, or clinical criteria.
Core may preferentially bind positive strand RNA, but no discrete encapsidation signal is apparent. By analogy to HBV pregenomic RNA, which binds to both of its gene products to facilitate packaging and replication, the HCV positive strand may bind core and NS5B cotranslationally. Binding to core might suppress IRES activity, leading to a transition from replication to assembly. Another event which may contribute to virion formation is the C-terminal proteolytic processing of the core and E2 proteins.
Forcione, MD, Raymond T. Chung, MD, and Jules L. Dienstag, MD CONTENTS INTRODUCTION ACUTE HBV INFECTION CHRONIC HEPATITIS B VIRUS INFECTION IMPACT OF IMMUNOPATHOGENETIC MECHANISMS OF HEPATITIS INJURY ON OUTCOME OF HEPATITIS B EXTRAHEPATIC MANIFESTATIONS IN CHRONIC HEPATITIS B VIRUS INFECTION PROGNOSIS AND SURVIVAL IN CHRONIC HEPATITIS B HCC AND CHRONIC HEPATITIS B VIRUS INFECTION INFECTION WITH HBV MUTANTS VACCINATION IN HBV INFECTION ANTIVIRAL THERAPY FOR CHRONIC HEPATITIS B VIRUS INFECTION ROLE OF LIVER TRANSPLANT IN HEPATITIS B FUTURE CONSIDERATIONS REFERENCES INTRODUCTION Hepatitis B virus (HBV) infects over 350 million persons, and represents one of the world’s leading public health problems.